1. Maternal exposure to environmental stress can modify progeny biology, but the mechanisms by which this occurs are not well understood.
2. C. elegans larvae can enter a state of immobile arrested development in response to osmotic stress.
3. Developmental arrest in response to osmotic stress is caused by the inhibition of insulin-like signalling and correlates with enhanced survival.
The article “Insulin-like Signalling to the Maternal Germline Controls Progeny Response to Osmotic Stress” is an informative and reliable source of information on how maternal exposure to environmental stress can protect progeny from future environmental stress. The article provides evidence that C. elegans larvae can enter a state of immobile arrested development in response to osmotic stress, and that this arrest is caused by the inhibition of insulin-like signalling and correlates with enhanced survival. The authors provide detailed descriptions of their experiments and results, as well as clear explanations for their conclusions, making it easy for readers to understand the implications of their findings.
The article does not appear to be biased or one-sided; it presents both sides equally and does not make any unsupported claims or omit any points of consideration or evidence for its claims. Furthermore, there are no promotional elements present in the article, nor does it appear partial in any way. The authors also note possible risks associated with their findings, such as potential deleterious effects on progeny due to maternal exposure to environmental stress.
In conclusion, this article is a trustworthy and reliable source of information on how maternal exposure to environmental stress can protect progeny from future environmental stress.